Frequently Asked Questions About Marijuana Usage in Colorado

 Today, marijuana is at the forefront of international controversy debating the appropriateness of its widespread illegal status. In many linkage states it has become legalized for medical purposes. This trend is known as "medical marijuana" and is strongly much-admired by advocates while simultaneously loathed coarsely by opponents (Dubner, 2007; Nakay, 2007; Van Tuyl, 2007). It is in this context that it was established to choose the subject of the inborn and pharmacological effects of marijuana for the basis of this research article.

What is marijuana?

Marijuana is a reforest more correctly called cannabis sativa. As mentioned, some cannabis sativa flora and fauna accomplish not have abuse potential and are called hemp. Hemp is used widely for various fiber products including newspaper and artist's canvas. Cannabis sativa with abuse potential is what we call marijuana (Doweiko, 2009). It is engaging to note that although widely studies for many years, there is a lot that researchers still reach not know not quite marijuana. Neuroscientists and biologists know what the effects of marijuana are but they still do not thoroughly understand why (Hazelden, 2005).

Deweiko (2009), Gold, Frost-Pineda, & Jacobs (2004) point out that of approximately four hundred known chemicals found in the cannabis plants, researchers know of exceeding sixty that are thought to have psychoactive effects upon the human brain. The most capably known and potent of these is -9-tetrahydrocannabinol, or THC. later than Hazelden (2005), Deweiko states that even though we know many of the neurophysical effects of THC, the reasons THC produces these effects are unclear.

Neurobiology:

As a psychoactive substance, THC directly affects the central aquiver system (CNS). It affects a omnipresent range of neurotransmitters and catalyzes supplementary biochemical and enzymatic ruckus as well. The CNS is stimulated like the THC activates specific neuroreceptors in the brain causing the various brute and emotional reactions that will be expounded on more specifically further on. The unaccompanied substances that can activate neurotransmitters are substances that mimic chemicals that the brain produces naturally. The fact that THC stimulates brain accomplish teaches scientists that the brain has natural cannabinoid receptors. It is still indistinct why humans have natural cannabinoid receptors and how they take steps (Hazelden, 2005; Martin, 2004). What we get know is that marijuana will breathing cannabinoid receptors going on to twenty period more actively than any of the body's natural neurotransmitters ever could (Doweiko, 2009).

Perhaps the biggest secrecy of all is the attachment in the middle of THC and the neurotransmitter serotonin. Serotonin receptors are accompanied by the most stimulated by every psychoactive drugs, but most specifically alcohol and nicotine. Independent of marijuana's membership with the chemical, serotonin is already a tiny understood neurochemical and its supposed neuroscientific roles of vigorous and intention are still mostly literary (Schuckit & Tapert, 2004). What neuroscientists have found definitively is that marijuana smokers have totally high levels of serotonin bustle (Hazelden, 2005). I would hypothesize that it may be this attachment amongst THC and serotonin that explains the "marijuana child maintenance program" of achieving abstinence from alcohol and allows marijuana smokers to avoid throbbing dissolution symptoms and avoid cravings from alcohol. The efficacy of "marijuana maintenance" for aiding alcohol abstinence is not scientific but is a phenomenon I have personally witnessed with numerous clients.

Interestingly, marijuana mimics hence many neurological reactions of new drugs that it is agreed hard to classify in a specific class. Researchers will area it in any of these categories: psychedelic; hallucinogen; or serotonin inhibitor. It has properties that mimic similar chemical responses as opioids. other chemical responses mimic stimulants (Ashton, 2001; Gold, Frost-Pineda, & Jacobs, 2004). Hazelden (2005) classifies marijuana in its own special class - cannabinoids. The defense for this confusion is the complexity of the numerous psychoactive properties found within marijuana, both known and unknown. One recent client I saying could not recover from the visual distortions he suffered as a consequences of pervasive psychedelic use as long as he was still smoking marijuana. This seemed to be as a consequences of the psychedelic properties found within sprightly cannabis (Ashton, 2001). Although not mighty passable to develop these visual distortions upon its own, marijuana was mighty passable to prevent the brain from healing and recovering.

Emotions:

Cannibinoid receptors are located throughout the brain appropriately affecting a wide variety of functioning. The most important upon the emotional level is the stimulation of the brain's nucleus accumbens perverting the brain's natural recompense centers. substitute is that of the amygdala which controls one's emotions and fears (Adolphs, Trane, Damasio, & Damaslio, 1995; Van Tuyl, 2007).

I have observed that the stuffy marijuana smokers who I fake like personally seem to ration a commonality of using the drug to control their anger. This observation has evidenced based outcome and is the basis of much scientific research. Research has in fact found that the link amongst marijuana and managing arouse is clinically significant (Eftekhari, Turner, & Larimer, 2004). anger is a reason mechanism used to protect against emotional upshot of adversity fueled by panic (Cramer, 1998). As stated, startle is a primary law controlled by the amygdala which is heavily stimulated by marijuana use (Adolphs, Trane, Damasio, & Damaslio, 1995; Van Tuyl, 2007).

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Neurophysical Effects of THC:

Neurological messages between transmitters and receptors not only manage emotions and psychological functioning. It is as well as how the body controls both volitional and nonvolitional functioning. The cerebellum and the basal ganglia run all swine doings and coordination. These are two of the most abundantly stimulated areas of the brain that are triggered by marijuana. This explains marijuana's physiological effect causing altered blood pressure (Van Tuyl, 2007), and a weakening of the muscles (Doweiko, 2009). THC ultimately affects all neuromotor commotion to some degree (Gold, Frost-Pineda, & Jacobs, 2004).

An fascinating phenomena I have witnessed in concerning every clients who identify marijuana as their drug of option is the use of marijuana smoking previously eating. This is explained by effects of marijuana upon the "CB-1" receptor. The CB-1 receptors in the brain are found heavily in the limbic system, or the nucleolus accumbens, which controls the return pathways (Martin, 2004). These recompense pathways are what feign the appetite and eating habits as allocation of the body's natural leftover instinct, causing us to crave eating food and rewarding us in the manner of dopamine considering we finally complete (Hazeldon, 2005). Martin (2004) makes this connection, pointing out that unique to marijuana users is the stimulation of the CB-1 receptor directly triggering the appetite